Dados consolidados até 30/06/2008.
Casos de aids identificados no Rio Grande do Sul
Ano Notificação: 2007
Município(Not): 430510 Caxias do Sul
Período: 2007
Ano Diagnóstico
Freqüência
TOTAL
76
2007
www.tabnet.datasus.gov.br
segunda-feira, 4 de maio de 2009
Os anos relacionados no campo "Períodos Disponíveis" são baseados no ano de diagnóstico.
Dados consolidados até 30/06/2008.
Casos de aids identificados no Rio Grande do Sul Freqüência segundo Ano Diagnóstico
Ano Notificação: 2007
Município(Not): 430510 Caxias do Sul
Período: 2007
Ano Diagnóstico
Freqüência
TOTAL
76
2007
www.tabnet.datasus.gov.br
Dados consolidados até 30/06/2008.
Casos de aids identificados no Rio Grande do Sul
Ano Notificação: 2007
Município(Not): 430510 Caxias do Sul
Período: 2007
Ano Diagnóstico
Freqüência
TOTAL
76
2007
www.tabnet.datasus.gov.br
O indice de morbidadeHospitalar do SUS
Morbidade Hospitalar do SUS
- por local de residência - Paraíba
Internações por Capítulo CID-10 segundo Município
Município: João Pessoa
Faixa Etária 2: Menor 1 ano
Período: Nov/2008-Fev/2009
Município
Fonte: Ministério da Saúde - Sistema de Informações Hospitalares do SUS (SIH/SUS)Nota: Dados preliminares atualizados em 17/04/2009, sujeitos a novas atualizações.Consulte o site da Secretaria Estadual de Saúde para mais informações.
TOTAL : populacao internada em Joao Pessoa:
250750 João Pessoa
Numero de morbidade hospitalar SUS:
731
Município: Recife
Causa - CID-BR-10: Tuberculose respiratória
Período: 2006
Município
Cap 01
Total
Fonte: MS/SVS/DASIS - Sistema de Informações sobre Mortalidade - SIMConsulte o site da Secretaria Estadual de Saúde para mais informações.
TOTAL
119
119
261160 Recife
119
119
Copia como .CSV
Copia para TabWin
www.tabnet.datasus.gov.br
segunda-feira, 13 de abril de 2009
Selecionar
texto integral
para imprimir
enviar por email
título:
Resolução RE nº 1760, de 03 de novembro de 2003
ementa não oficial:
Concede registro de risco II, revalidação(ões) de registro alteração(ões) de rotulagem, alteração(ões)/inclusão(ões) de destinação de uso e reconsideração(ões) de indeferimento de produtos Saneantes Domissanitários
publicação:
D.O.U. - Diário Oficial da União; Poder Executivo, de 04 de novembro de 2003
órgão emissor:
ANVISA - Agência Nacional de Vigilância Sanitária
alcance do ato:
federal - Brasil
área de atuação:
Saneantes
2/2
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título:
Decreto nº 23430, de 24 de outubro de 1974
ementa:
Aprova Regulamento que dispõe sobre a promoção, proteção e recuperação da Saúde Pública.
publicação:
D.O.E. - Diário Oficial do Estado, 1974
órgão emissor:
Governo do Estado
alcance do ato:
estadual - Porto Alegre / Rio Grande do Sul / Brasil
área de atuação:
Tecnologia de Serviços de Saúde
Art. 118 revogada(o) por: Decreto nº 30191, de 15 de junho de 1981
relacionamento(s):
revoga:
Decreto nº 14196, de 04 de outubro de 1962
Decreto nº 7558, de 11 de novembro de 1938
www.anvisa.gov.br
texto integral
para imprimir
enviar por email
título:
Resolução RE nº 1760, de 03 de novembro de 2003
ementa não oficial:
Concede registro de risco II, revalidação(ões) de registro alteração(ões) de rotulagem, alteração(ões)/inclusão(ões) de destinação de uso e reconsideração(ões) de indeferimento de produtos Saneantes Domissanitários
publicação:
D.O.U. - Diário Oficial da União; Poder Executivo, de 04 de novembro de 2003
órgão emissor:
ANVISA - Agência Nacional de Vigilância Sanitária
alcance do ato:
federal - Brasil
área de atuação:
Saneantes
2/2
Selecionar
texto integral
para imprimir
enviar por email
título:
Decreto nº 23430, de 24 de outubro de 1974
ementa:
Aprova Regulamento que dispõe sobre a promoção, proteção e recuperação da Saúde Pública.
publicação:
D.O.E. - Diário Oficial do Estado, 1974
órgão emissor:
Governo do Estado
alcance do ato:
estadual - Porto Alegre / Rio Grande do Sul / Brasil
área de atuação:
Tecnologia de Serviços de Saúde
Art. 118 revogada(o) por: Decreto nº 30191, de 15 de junho de 1981
relacionamento(s):
revoga:
Decreto nº 14196, de 04 de outubro de 1962
Decreto nº 7558, de 11 de novembro de 1938
www.anvisa.gov.br
segunda-feira, 30 de março de 2009
GENERAL METABOLISM BACTERIAL
Medical Microbiology Section 1. Bacteriology4. Bacterial MetabolismPeter Jurtshuk Jr. General ConceptsHeterotrophic Metabolism
Heterotrophic metabolism is the biologic oxidation of organic compounds, such as glucose, to yield ATP and simpler organic (or inorganic) compounds, which are needed by the bacterial cell for biosynthetic or assimilatory reactions.Respiration
Respiration is a type of heterotrophic metabolism that uses oxygen and in which 38 moles of ATP are derived from the oxidation of 1 mole of glucose, yielding 380,000 cal. (An additional 308,000 cal is lost as heat.)Fermentation
In fermentation, another type of heterotrophic metabolism, an organic compound rather than oxygen is the terminal electron (or hydrogen) acceptor. Less energy is generated from this incomplete form of glucose oxidation, but the process supports anaerobic growth.Krebs Cycle
The Krebs cycle is the oxidative process in respiration by which pyruvate (via acetyl coenzyme A) is completely decarboxylated to CO2. The pathway yields 15 moles of ATP (150,000 calories).Glyoxylate Cycle
The glyoxylate cycle, which occurs in some bacteria, is a modification of the Krebs cycle. Acetyl coenzyme A is generated directly from oxidation of fatty acids or other lipid compounds.Electron Transport and Oxidative Phosphorylation
In the final stage of respiration, ATP is formed through a series of electron transfer reactions within the cytoplasmic membrane that drive the oxidative phosphorylation of ADP to ATP. Bacteria use various flavins, cytochrome, and non-heme iron components as well as multiple cytochrome oxidases for this process.Mitchell or Proton Extrusion Hypothesis
The Mitchell hypothesis explains the energy conservation in all cells on the basis of the selective extrusion of H+ ions across a proton-impermeable membrane, which generates a proton motive force. This energy allows for ATP synthesis both in respiration and photosynthesis.Bacterial Photosynthesis
Bacterial photosynthesis is a light-dependent, anaerobic mode of metabolism. Carbon dioxide is reduced to glucose, which is used for both biosynthesis and energy production. Depending on the hydrogen source used to reduce CO2, both photolithotrophic and photoorganotrophic reactions exist in bacteria.Autotrophy
Autotrophy is a unique form of metabolism found only in bacteria. Inorganic compounds are oxidized directly (without using sunlight) to yield energy (e.g., NH3, NO2–, S2, and Fe2+). This metabolic mode also requires energy for CO2 reduction, like photosynthesis, but no lipid-mediated processes are involved. This metabolic mode has also been called chemotrophy, chemoautotrophy, or chemolithotrophy.Anaerobic Respiration
Anaerobic respiration is another heterotrophic mode of metabolism in which a specific compound other than O2 serves as a terminal electron acceptor. Such acceptor compounds include NO3–, SO42–, fumarate, and even CO2 for methane-producing bacteria.The Nitrogen Cycle
The nitrogen cycle consists of a recycling process by which organic and inorganic nitrogen compounds are used metabolically and recycled among bacteria, plants, and animals. Important processes, including ammonification, mineralization, nitrification, denitrification, and nitrogen fixation, are carried out primarily by bacteria.
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© The University of Texas Medical Branch at Galveston
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Heterotrophic metabolism is the biologic oxidation of organic compounds, such as glucose, to yield ATP and simpler organic (or inorganic) compounds, which are needed by the bacterial cell for biosynthetic or assimilatory reactions.Respiration
Respiration is a type of heterotrophic metabolism that uses oxygen and in which 38 moles of ATP are derived from the oxidation of 1 mole of glucose, yielding 380,000 cal. (An additional 308,000 cal is lost as heat.)Fermentation
In fermentation, another type of heterotrophic metabolism, an organic compound rather than oxygen is the terminal electron (or hydrogen) acceptor. Less energy is generated from this incomplete form of glucose oxidation, but the process supports anaerobic growth.Krebs Cycle
The Krebs cycle is the oxidative process in respiration by which pyruvate (via acetyl coenzyme A) is completely decarboxylated to CO2. The pathway yields 15 moles of ATP (150,000 calories).Glyoxylate Cycle
The glyoxylate cycle, which occurs in some bacteria, is a modification of the Krebs cycle. Acetyl coenzyme A is generated directly from oxidation of fatty acids or other lipid compounds.Electron Transport and Oxidative Phosphorylation
In the final stage of respiration, ATP is formed through a series of electron transfer reactions within the cytoplasmic membrane that drive the oxidative phosphorylation of ADP to ATP. Bacteria use various flavins, cytochrome, and non-heme iron components as well as multiple cytochrome oxidases for this process.Mitchell or Proton Extrusion Hypothesis
The Mitchell hypothesis explains the energy conservation in all cells on the basis of the selective extrusion of H+ ions across a proton-impermeable membrane, which generates a proton motive force. This energy allows for ATP synthesis both in respiration and photosynthesis.Bacterial Photosynthesis
Bacterial photosynthesis is a light-dependent, anaerobic mode of metabolism. Carbon dioxide is reduced to glucose, which is used for both biosynthesis and energy production. Depending on the hydrogen source used to reduce CO2, both photolithotrophic and photoorganotrophic reactions exist in bacteria.Autotrophy
Autotrophy is a unique form of metabolism found only in bacteria. Inorganic compounds are oxidized directly (without using sunlight) to yield energy (e.g., NH3, NO2–, S2, and Fe2+). This metabolic mode also requires energy for CO2 reduction, like photosynthesis, but no lipid-mediated processes are involved. This metabolic mode has also been called chemotrophy, chemoautotrophy, or chemolithotrophy.Anaerobic Respiration
Anaerobic respiration is another heterotrophic mode of metabolism in which a specific compound other than O2 serves as a terminal electron acceptor. Such acceptor compounds include NO3–, SO42–, fumarate, and even CO2 for methane-producing bacteria.The Nitrogen Cycle
The nitrogen cycle consists of a recycling process by which organic and inorganic nitrogen compounds are used metabolically and recycled among bacteria, plants, and animals. Important processes, including ammonification, mineralization, nitrification, denitrification, and nitrogen fixation, are carried out primarily by bacteria.
Download this book for your handheld computer (requires the MobiPocket Reader).
© The University of Texas Medical Branch at Galveston
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segunda-feira, 23 de março de 2009
Pritchett CL, Jones AK, Carterson AJ, Jackson D, Frisk A, Wolfgang MC, Schurr MJ.
University of Colorado Denver School of Medicine, Department of Microbiology, Aurora, CO 80045; Department of Microbiology and Immunology, University of North Carolina, Chapel Hill, NC 27599; Department of Microbiology and Immunology, Tulane University School of Medicine, New Orleans, LA, 70112; Department of Biology, University of Louisiana at Monroe, LA 71209; Cystic Fibrosis/Pulmonary Research and Treatment Center, University of North Carolina, Chapel Hill, NC 27599.
Pseudomonas aeruginosa is an opportunistic pathogen that causes chronic infections in individuals suffering from the genetic disorder cystic fibrosis. In P. aeruginosa, the transcriptional regulator AlgR controls a variety of virulence factors, including alginate production, twitching motility, biofilm formation, quorum sensing and hydrogen cyanide (HCN) production. In this study, the regulation of HCN production was examined. Strains lacking AlgR or the putative AlgR sensor, AlgZ, produced significantly less HCN compared to a non-mucoid isogenic parent. In contrast, algR and algZ mutants showed increased HCN production in an alginate producing (mucoid) background. HCN production was optimal in a 5% O2 environment. In addition, cyanide production was elevated in bacteria grown on an agar surface as compared to the same bacteria grown in planktonic culture. A conserved AlgR phosphorylation site (aspartate at amino acid position 54), which is required for surface-dependent twitching motility, but not alginate production, was found to be critical for cyanide production. Nuclease protection mapping of the hcnA promoter identified a new transcriptional start site required for HCN production. A subset of clinical isolates that lack this start site produced low amounts of cyanide. Taken together, these data show that the P. aeruginosa hcnA promoter contains multiple transcriptional start sites, that HCN production is regulated by AlgZ and AlgR and maximal under microaerobic conditions when the organism is surface attached.
PMID: 19270096 [PubMed - as supplied by publisher]
University of Colorado Denver School of Medicine, Department of Microbiology, Aurora, CO 80045; Department of Microbiology and Immunology, University of North Carolina, Chapel Hill, NC 27599; Department of Microbiology and Immunology, Tulane University School of Medicine, New Orleans, LA, 70112; Department of Biology, University of Louisiana at Monroe, LA 71209; Cystic Fibrosis/Pulmonary Research and Treatment Center, University of North Carolina, Chapel Hill, NC 27599.
Pseudomonas aeruginosa is an opportunistic pathogen that causes chronic infections in individuals suffering from the genetic disorder cystic fibrosis. In P. aeruginosa, the transcriptional regulator AlgR controls a variety of virulence factors, including alginate production, twitching motility, biofilm formation, quorum sensing and hydrogen cyanide (HCN) production. In this study, the regulation of HCN production was examined. Strains lacking AlgR or the putative AlgR sensor, AlgZ, produced significantly less HCN compared to a non-mucoid isogenic parent. In contrast, algR and algZ mutants showed increased HCN production in an alginate producing (mucoid) background. HCN production was optimal in a 5% O2 environment. In addition, cyanide production was elevated in bacteria grown on an agar surface as compared to the same bacteria grown in planktonic culture. A conserved AlgR phosphorylation site (aspartate at amino acid position 54), which is required for surface-dependent twitching motility, but not alginate production, was found to be critical for cyanide production. Nuclease protection mapping of the hcnA promoter identified a new transcriptional start site required for HCN production. A subset of clinical isolates that lack this start site produced low amounts of cyanide. Taken together, these data show that the P. aeruginosa hcnA promoter contains multiple transcriptional start sites, that HCN production is regulated by AlgZ and AlgR and maximal under microaerobic conditions when the organism is surface attached.
PMID: 19270096 [PubMed - as supplied by publisher]
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